Rain Therapeutics to Participate in Citi’s 17th Annual BioPharma Conference
Rain Therapeutics (NasdaqGS: RAIN) has announced its participation in Citi’s 17th Annual BioPharma Conference, scheduled for September 7-8, 2022, at the Four Seasons Hotel, Boston, MA. CEO Avanish Vellanki will be part of a targeted oncology panel on September 8 from 2:40-3:25 PM ET. The company focuses on developing precision oncology therapies, including milademetan, a small molecule inhibitor of the MDM2-p53 complex, which reactivates p53. Rain emphasizes a tumor-agnostic strategy to select patients based on genetic profiles.
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NEWARK, Calif., Aug. 31, 2022 (GLOBE NEWSWIRE) -- Rain Therapeutics Inc. (NasdaqGS: RAIN), (“Rain”), a late-stage company developing precision oncology therapeutics with a lead product candidate, milademetan, an oral, small molecule inhibitor of the MDM2-p53 complex that reactivates p53, today announced that company management will be participating in Citi’s 17th Annual BioPharma Conference on September 7-8, 2022 taking place at the Four Seasons Hotel in Boston, MA.
Company management will participate in one-on-one investor meetings, and Avanish Vellanki, CEO, will be participating in a company panel for targeted oncology on Thursday, September 8 at 2:40-3:25 PM ET. The webcast of the panel is linked here.
If you would like to request a meeting with management, please contact your Citi representative.
About Rain Therapeutics Inc.
Rain Therapeutics Inc. is a late-stage precision oncology company developing therapies that target oncogenic drivers for which it is able to genetically select patients it believes will most likely benefit. This approach includes using a tumor-agnostic strategy to select patients based on their tumors’ underlying genetics rather than histology. Rain’s lead product candidate, milademetan, is a small molecule, oral inhibitor of MDM2-p53 complex that reactivates p53. In addition to milademetan, Rain is also developing a preclinical program that is focused on inducing synthetic lethality in cancer cells by inhibiting RAD52.
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