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IDEAYA Biosciences Announces IDE892, a Potential Best-in-Class MTA-Cooperative PRMT5 Inhibitor, Initiates a Phase 1/2 Clinical Combination Study in MTAP-Deleted Pancreatic and Lung Cancers

(Positive)

IDEAYA Biosciences (NASDAQ: IDYA) enrolled the first patient in a Phase 1/2 trial of IDE892, a potential best-in-class MTA-cooperative PRMT5 inhibitor, combined with IDE397 (MAT2A inhibitor) in MTAP-deleted solid tumors, focusing on pancreatic and non-small cell lung cancer.

IDE892 showed favorable CYP450 properties, multiple cleared dose cohorts with no maximum tolerated dose reached, and monotherapy expansion is anticipated in Q3 2026. IDEAYA also announced a Roche collaboration and a third MTAP-deleted program targeting CDKN2A.

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AI-generated analysis. How Rhea-AI works. Not financial advice.

Positive

  • First patient enrolled in Phase 1/2 IDE892 + IDE397 MTAP-deleted trial
  • IDE892 Phase 1 monotherapy cleared multiple dose cohorts, MTD not yet reached
  • IDE892 shows CYP3A4 IC50 >45 µM and no time-dependent inhibition of 7 CYPs
  • Monotherapy expansion of IDE892 anticipated in Q3 2026
  • Clinical collaboration with Roche combining IDE892 with RG6505 pan-RAS inhibitor in MTAP-deleted PDAC
  • Third MTAP-deleted program targeting CDKN2A aiming for IND in first half of 2027

Negative

  • None.

News Market Reaction – IDYA

+4.31%
1 alert
+4.31% News Effect
+$112M Valuation Impact
$2.71B Market Cap
0.0x Rel. Volume

On the day this news was published, IDYA gained 4.31%, reflecting a moderate positive market reaction. This price movement added approximately $112M to the company's valuation, bringing the market cap to $2.71B at that time.

Data tracked by StockTitan Argus on the day of publication.

Market Context

This announcement extends IDEAYA’s MTAP-deleted strategy by combining IDE892, an MTA‑cooperative PRM...
Analysis

This announcement extends IDEAYA’s MTAP-deleted strategy by combining IDE892, an MTA‑cooperative PRMT5 inhibitor, with IDE397 in a Phase 1/2 trial for pancreatic and lung cancers, where MTAP loss occurs in up to 40% and 15–20% of cases, respectively. It complements prior darovasertib advances and the Roche collaboration. Investors may watch for IDE892 safety, early efficacy signals, and timing of the planned IDE892 expansion in Q3 2026 and the CDKN2A IND in 1H 2027.

Key Figures

CYP3A4 IC50: >45 micromolar MTA-PRMT5 selectivity: 1,400-fold Pancreatic MTAP deletion: up to 40% +4 more
7 metrics
CYP3A4 IC50 >45 micromolar IDE892 CYP3A4 inhibition threshold
MTA-PRMT5 selectivity 1,400-fold Selective MTA-PRMT5 vs SAM-PRMT5 cooperative binding for IDE892
Pancreatic MTAP deletion up to 40% Estimated MTAP-deletion rate in pancreatic cancer
NSCLC MTAP deletion 15–20% Estimated MTAP-deletion rate in non-small cell lung cancer
Solid tumor MTAP deletion approximately 15% Estimated MTAP-deletion rate across all solid tumors
IDE892 expansion timing Q3 2026 Anticipated IDE892 monotherapy Phase 1 expansion start
CDKN2A IND target 1H 2027 Planned IND application timing for MTAP-deleted CDKN2A program

Previous Clinical trial Reports

5 past events · Latest: Jun 01 (Positive)
Same Type Pattern 5 events
Date Event Sentiment 24h Move Catalyst
Jun 01 Registrational data update Positive -1.8% Reported complete Phase 2/3 darovasertib + crizotinib data in uveal melanoma.
Apr 30 RTOR filing plan Positive +3.7% Planned FDA RTOR submission for darovasertib combo after positive topline data.
Apr 21 ASCO data presentation Neutral -5.3% Announced late-breaking ASCO oral presentation for complete OptimUM-02 trial data.
Apr 13 Topline trial results Positive +7.6% Released positive Phase 2/3 topline darovasertib + crizotinib results in uveal melanoma.
Apr 06 First-patient-in trial Positive -1.2% Announced first-patient-in for Phase 1 IDE574 KAT6/7 inhibitor solid tumor trial.

24h Move is the share-price change in the day after each event; other market factors may also have contributed.

Pattern Detected

Clinical headlines have produced mixed reactions: strong positive Phase 2/3 data and RTOR steps often aligned with gains, while other clinically positive updates sometimes saw modest sell-offs.

Recent Company History

Over the last few months, IDEAYA’s pipeline has advanced through multiple clinical milestones. Key events include positive Phase 2/3 darovasertib + crizotinib data with improved PFS and ORR on Apr 13, 2026, subsequent ASCO presentation plans, and an FDA RTOR/NDA path. The company also initiated a first‑patient‑in Phase 1 trial for IDE574 in solid tumors. Today’s IDE892/IDE397 MTAP‑deleted study fits this pattern of broadening precision-oncology combinations beyond uveal melanoma.

Key Terms

prmt5, mat2a, pan-ras inhibitor, mtap-deleted, +2 more
6 terms
prmt5 medical
"a potential best-in-class methylthioadenosine (MTA)-cooperative inhibitor of PRMT5, in combination"
PRMT5 is a cellular enzyme that adds small chemical tags to specific spots on other proteins, which changes how those proteins behave and how genes are read. Investors care because blocking or modifying PRMT5 can alter cell growth and survival, making it a promising drug target—especially in cancer—so clinical trial results, regulatory decisions, or biomarker developments related to PRMT5 programs can materially affect the value and risk of companies working in that space.
mat2a medical
"inhibitor of PRMT5, in combination with IDE397, a potential first-in-class and best-in-class inhibitor of MAT2A, in MTAP-deleted"
MAT2A is a gene that makes an enzyme responsible for producing a key cellular chemical (S‑adenosylmethionine) used to add small chemical “tags” that control gene activity and cell growth. For investors, MAT2A matters because drugs that block or modulate this enzyme act like a control knob for diseased cells—successful therapies can drive significant company value, while development and approval risks mean outcomes are uncertain.
pan-ras inhibitor medical
"clinical collaboration with Roche evaluating IDE892 in combination with RG6505, Roche's Phase 1 pan-RAS inhibitor, in MTAP-deleted"
A pan-RAS inhibitor is a drug designed to block activity of the RAS family of proteins (such as KRAS, NRAS and HRAS) that often drive uncontrolled cell growth in cancers. For investors, it matters because a single medicine that works across multiple RAS types can address a larger group of patients and reduce the risk that a tumor will bypass the drug, much like cutting power to several faulty switches instead of just one.
mtap-deleted medical
"Phase 1 clinical trial evaluating IDE892 ... in MTAP-deleted solid tumors, with a focus on NSCLC and pancreatic cancer."
mtap-deleted describes cells or tumors that have lost the MTAP gene, a molecular ‘tool’ normally involved in a basic cellular recycling pathway. For investors, this matters because that missing tool can create a specific weakness drug developers can target, helping identify which patients a therapy might work for and shaping clinical trial design, potential market size, and the commercial value of precision medicines.
cytochrome p450s medical
"did not show time dependent inhibition of any of the 7 major cytochrome P450s (CYP1A2, CYP2B6, CYP2C8, CYP2C9, CYP2C19, CYP2D6, CYP3A4)"
Cytochrome P450s are a large family of enzymes, mainly in the liver, that chemically modify drugs, toxins and hormones so the body can eliminate them—think of them as molecular scissors or a processing plant that changes incoming substances before disposal. Their activity determines how fast a medicine is cleared, how strong its effects or side effects may be, and whether it will interact with other drugs, so they are crucial for assessing a drug candidate’s safety, dosing and regulatory risk.
ind application regulatory
"preclinical toxicology studies to support an investigational new drug (IND) application in the first half of 2027."
An Investigational New Drug (IND) application is a formal request to regulators to allow a drug or biologic to be tested in people. It bundles lab and animal safety data, manufacturing details, and a plan for human trials—think of it like a building permit that lets developers move from design to construction. For investors, IND approval is a key milestone that permits clinical testing, reduces regulatory uncertainty, and can materially affect a company’s timeline, risk profile, and valuation.

AI-generated analysis. How Rhea-AI works. Not financial advice.

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  • IDE892 is a potential best-in-class MTA-Cooperative PRMT5 combination partner with MAT2A and pan-RAS inhibitors, with favorable drug-like properties
  • IDE892 has a CYP3A4 IC50 greater than 45 micromolar, and did not show time dependent inhibition of any of the 7 major cytochrome P450s (CYP1A2, CYP2B6, CYP2C8, CYP2C9, CYP2C19, CYP2D6, CYP3A4) based on full kinetic CYP inactivation assays
  • IDE892 monotherapy Phase 1 escalation has cleared multiple dose cohorts with maximally efficacious target exposures anticipated at a favorable pill size and the MTD has not yet been reached. IDE892 monotherapy expansion is anticipated in Q3 2026
  • MTAP-deletion is estimated to occur in up to 40% of pancreatic cancer and ~15% of non-small cell lung cancer (NSCLC)

SOUTH SAN FRANCISCO, Calif., June 15, 2026 /PRNewswire/ -- IDEAYA Biosciences, Inc. (NASDAQ: IDYA), a leading precision medicine oncology company, today announced that the first patient has been enrolled in its Phase 1 clinical trial evaluating IDE892, a potential best-in-class methylthioadenosine (MTA)-cooperative inhibitor of PRMT5, in combination with IDE397, a potential first-in-class and best-in-class inhibitor of MAT2A, in MTAP-deleted solid tumors, with a focus on NSCLC and pancreatic cancer. In preclinical studies, dual inhibition of PRMT5 and MAT2A with the combination of IDE892 and IDE397 resulted in potent anti-tumor activity in MTAP-deleted tumor models, including complete and durable responses at well-tolerated doses below those required for monotherapy activity.

"We are excited to begin enrolling this Phase 1 combination trial evaluating IDE892 in MTAP-deleted pancreatic cancer and non-small cell lung cancer. We designed IDE892 with potential best-in-class properties, including approximately 1,400-fold selective MTA-PRMT5 cooperative binding versus SAM-PRMT5 cooperative binding intended to maximize its therapeutic window and favorable drug-like properties to enable rational combinations with IDE397 and pan-RAS inhibitors. This trial exemplifies our clinical development strategy of enabling rational combinations to deliver deeper and more durable responses for MTAP-deleted pancreatic cancer and lung cancer patients where there are currently no approved treatment options," said Yujiro S. Hata, President and Chief Executive Officer, IDEAYA Biosciences.

Loss of MTAP leads to the accumulation of MTA and increased dependence on PRMT5 and MAT2A, two key enzymes involved in methylation and RNA splicing. In MTAP-deleted tumors, this biology establishes a robust synthetic lethal vulnerability that underpins the mechanistic rationale for combining IDE892 and IDE397. IDEAYA also entered into a clinical collaboration with Roche evaluating IDE892 in combination with RG6505, Roche's Phase 1 pan-RAS inhibitor, in MTAP-deleted pancreatic ductal adenocarcinoma (PDAC) to target the genetic co-alterations of MTAP and KRAS in this indication. Next, IDEAYA is advancing a third proprietary program for MTAP-deleted solid tumors targeting CDKN2A, the most common co-alteration of MTAP, through ongoing preclinical toxicology studies to support an investigational new drug (IND) application in the first half of 2027.

MTAP deletion is estimated to occur in approximately 15% of all solid tumors, including 15-20% of NSCLC and up to 40% of pancreatic cancer. There are no approved therapies for MTAP-deleted cancers, highlighting the significant unmet need and opportunity for new precision therapies for these patients.

About IDEAYA Biosciences

IDEAYA is a precision medicine oncology company committed to the discovery, development, and commercialization of transformative therapies for cancer. Our approach integrates expertise in small-molecule drug discovery, structural biology and bioinformatics with robust internal capabilities in identifying and validating translational biomarkers to develop tailored, potentially first-in-class targeted therapies aligned to the genetic drivers of disease. We have built a deep pipeline of product candidates focused on synthetic lethality and antibody-drug conjugates, or ADCs, for molecularly defined solid tumor indications. Our mission is to bring forth the next wave of precision oncology therapies that are more selective, more effective, and deeply personalized with the goal of altering the course of disease and improving clinical outcomes for patients with cancer.

Forward-Looking Statements

This press release contains forward-looking statements, including, but not limited to, statements related to the potential therapeutic benefits, safety, efficacy, tolerability, therapeutic window, selectivity, drug-like properties and best-in-class potential of IDE892; the potential of IDE892 as a combination partner with IDE397, RG6505 and other therapies; the anticipated enrollment, conduct, progress and timing of IDEAYA's Phase 1 clinical trials evaluating IDE892 as monotherapy and in combination with IDE397 and RG6505; expectations regarding the achievement of maximally efficacious target exposures, favorable pill size, dose escalation, maximum tolerated dose and clinical expansion cohorts; the potential for IDE892 and IDE397 to produce deeper, more durable or improved anti-tumor responses; the translational relevance of preclinical data and the ability of such data to predict future clinical outcomes; the prevalence of MTAP deletion in various tumor types and the associated market opportunity; the potential therapeutic utility of targeting MTAP-deleted cancers and related synthetic lethal vulnerabilities; the anticipated benefits of IDEAYA's collaboration with Roche; the advancement of IDEAYA's CDKN2A-targeted program and the anticipated timing of IND-enabling activities and regulatory submissions. Such forward-looking statements are based on management's current expectations, assumptions and beliefs and involve substantial risks and uncertainties that could cause actual results, including, but not limited to, those related to IDEAYA's clinical programs, commercial activities, and performance and/or achievements, to differ significantly and/or materially from those expressed or implied by the forward-looking statements. Such risks and uncertainties include, among others, the uncertainties inherent in the drug development process, including the process of designing and conducting preclinical and clinical trials, enrollment rates, safety outcomes, efficacy results, regulatory interactions and decisions, and the ability to translate preclinical findings into clinical benefit, manufacturing and supply risks, competition, changes in standard of care, the timing and success of commercialization efforts, the outcome of collaborations and licensing arrangements, IDEAYA's ability to successfully establish, protect and defend its intellectual property, and other matters that could affect the sufficiency of financial resources to fund operations. IDEAYA undertakes no obligation to update or revise any forward-looking statements. A further description of risks and uncertainties that could cause actual results to differ from those expressed in these forward-looking statements, as well as risks relating to the business of IDEAYA in general, are in IDEAYA's filings with the Securities and Exchange Commission, including IDEAYA's most recent Annual Report on Form 10-K and any current and periodic reports filed with the U.S. Securities and Exchange Commission.

Investor and Media Contact

IDEAYA Biosciences
Joshua Bleharski, Ph.D.
Chief Financial Officer
investor@ideayabio.com

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SOURCE IDEAYA Biosciences, Inc.

FAQ

What did IDEAYA Biosciences (NASDAQ: IDYA) announce about the IDE892 clinical trial on June 15, 2026?

IDEAYA announced first patient enrollment in a Phase 1/2 trial of IDE892 plus IDE397 for MTAP-deleted solid tumors. According to IDEAYA, the study focuses on MTAP-deleted pancreatic and non-small cell lung cancers and evaluates the dual inhibition of PRMT5 and MAT2A.

What is IDE892 and why is it important for MTAP-deleted cancers in IDYA's pipeline?

IDE892 is described as a potential best-in-class MTA-cooperative PRMT5 inhibitor for MTAP-deleted tumors. According to IDEAYA, it shows approximately 1,400-fold selective MTA-PRMT5 cooperative binding versus SAM-PRMT5, aiming to maximize therapeutic window and enable rational combinations with IDE397 and pan-RAS inhibitors.

How advanced is the IDE892 monotherapy program in IDEAYA Biosciences' (IDYA) clinical development?

IDE892 monotherapy is in Phase 1 dose escalation and has cleared multiple dose cohorts. According to IDEAYA, maximally efficacious target exposures are anticipated at a favorable pill size, the maximum tolerated dose has not yet been reached, and monotherapy expansion is expected in Q3 2026.

What is the collaboration between IDEAYA Biosciences (IDYA) and Roche involving IDE892?

IDEAYA entered a clinical collaboration with Roche to evaluate IDE892 with RG6505, Roche's Phase 1 pan-RAS inhibitor, in MTAP-deleted pancreatic ductal adenocarcinoma. According to IDEAYA, the combination targets co-alterations of MTAP and KRAS in this pancreatic cancer subtype.

How common are MTAP-deleted cancers targeted by IDEAYA's IDE892 program?

MTAP deletion is estimated in about 15% of all solid tumors, including 15–20% of NSCLC and up to 40% of pancreatic cancers. According to IDEAYA, there are currently no approved therapies for MTAP-deleted cancers, indicating a significant unmet medical need.

What other MTAP-deleted programs besides IDE892 is IDEAYA Biosciences (IDYA) developing?

IDEAYA is advancing a third proprietary MTAP-deleted solid tumor program targeting CDKN2A, the most common MTAP co-alteration. According to IDEAYA, ongoing preclinical toxicology studies aim to support an investigational new drug application in the first half of 2027.